Matches in SemOpenAlex for { <https://semopenalex.org/work/W2019292712> ?p ?o ?g. }
- W2019292712 endingPage "85" @default.
- W2019292712 startingPage "79" @default.
- W2019292712 abstract "The lesion patterns and mechanisms of ischemic stroke caused by extracranial internal carotid artery (EICA) stenosis are well understood. The prognosis of intracranial internal carotid artery (IICA) stenosis is very poor. However, little is known about lesion patterns and mechanisms of cerebral infarcts caused by IICA stenosis. The objective of this study was to investigate the lesion patterns and mechanisms of infarcts produced by severe IICA stenosis and compare it with that produced by severe EICA stenosis.We recruited 62 patients with acute cerebral infarcts who fulfilled the following criteria: 1) lesions of acute infarcts were verified by diffusion-weighted imaging (DWI) performed within 1 week of stroke onset; 2) infarct lesions were located within the territory of unilateral internal carotid artery (ICA); 3) infarcts were definitely caused by severe atherosclerosis stenosis (stenosis rate of 70%-99%) of the ipsilateral IICA or EICA, with all other potential causes of stroke being entirely excluded. According to the distributions of infarct lesions in the arterial territory of the ICA displayed on DWI, lesion patterns were classified as either 1) single infarct [perforating artery infarct (PAI), pial artery infarct (PI) or border-zone infarct (BZI)], or 2) multiple infarcts (a combination of types described above).There were 29 patients with ischemic stroke caused by severe IICA stenosis, and 33 patients with stroke caused by severe EICA stenosis. Single BZI (14/29, P=0.015), and infarcts involving the border zone (19/29, P=0.021) or the internal border zone (13/29, P=0.013) were identified more often in patients with IICA stenosis compared to those with EICA stenosis. PI and/or PAI (22/32, P=0.021) were identified more often in patients with EICA stenosis.Lesion patterns in patients with severe IICA stenosis were different from those with severe EICA stenosis. The hypoperfusion mechanism leading to BZI was more important for patients with severe IICA stenosis than for those with severe EICA stenosis. Embolisms leading to PI and/or PAI were more important for patients with severe EICA stenosis." @default.
- W2019292712 created "2016-06-24" @default.
- W2019292712 creator A5020078006 @default.
- W2019292712 creator A5023894502 @default.
- W2019292712 creator A5025564811 @default.
- W2019292712 creator A5030218520 @default.
- W2019292712 creator A5034129006 @default.
- W2019292712 creator A5043004258 @default.
- W2019292712 creator A5045414239 @default.
- W2019292712 creator A5078372604 @default.
- W2019292712 date "2011-08-01" @default.
- W2019292712 modified "2023-10-15" @default.
- W2019292712 title "Lesion patterns and mechanism of cerebral infarction caused by severe atherosclerotic intracranial internal carotid artery stenosis" @default.
- W2019292712 cites W1985759654 @default.
- W2019292712 cites W2001738836 @default.
- W2019292712 cites W2008483626 @default.
- W2019292712 cites W2022654399 @default.
- W2019292712 cites W2023593820 @default.
- W2019292712 cites W2029089798 @default.
- W2019292712 cites W2032503729 @default.
- W2019292712 cites W2035572126 @default.
- W2019292712 cites W2039801270 @default.
- W2019292712 cites W2041268528 @default.
- W2019292712 cites W2045545178 @default.
- W2019292712 cites W2049935780 @default.
- W2019292712 cites W2052972364 @default.
- W2019292712 cites W2075584356 @default.
- W2019292712 cites W2089356165 @default.
- W2019292712 cites W2090867081 @default.
- W2019292712 cites W2096605777 @default.
- W2019292712 cites W2100300473 @default.
- W2019292712 cites W2115891513 @default.
- W2019292712 cites W2131759134 @default.
- W2019292712 cites W2133341909 @default.
- W2019292712 cites W2138595885 @default.
- W2019292712 cites W2158381538 @default.
- W2019292712 cites W2167806412 @default.
- W2019292712 cites W2168139815 @default.
- W2019292712 cites W2767911074 @default.
- W2019292712 cites W4320288045 @default.
- W2019292712 doi "https://doi.org/10.1016/j.jns.2011.05.012" @default.
- W2019292712 hasPubMedId "https://pubmed.ncbi.nlm.nih.gov/21621797" @default.
- W2019292712 hasPublicationYear "2011" @default.
- W2019292712 type Work @default.
- W2019292712 sameAs 2019292712 @default.
- W2019292712 citedByCount "25" @default.
- W2019292712 countsByYear W20192927122012 @default.
- W2019292712 countsByYear W20192927122014 @default.
- W2019292712 countsByYear W20192927122015 @default.
- W2019292712 countsByYear W20192927122016 @default.
- W2019292712 countsByYear W20192927122017 @default.
- W2019292712 countsByYear W20192927122018 @default.
- W2019292712 countsByYear W20192927122019 @default.
- W2019292712 countsByYear W20192927122021 @default.
- W2019292712 countsByYear W20192927122023 @default.
- W2019292712 crossrefType "journal-article" @default.
- W2019292712 hasAuthorship W2019292712A5020078006 @default.
- W2019292712 hasAuthorship W2019292712A5023894502 @default.
- W2019292712 hasAuthorship W2019292712A5025564811 @default.
- W2019292712 hasAuthorship W2019292712A5030218520 @default.
- W2019292712 hasAuthorship W2019292712A5034129006 @default.
- W2019292712 hasAuthorship W2019292712A5043004258 @default.
- W2019292712 hasAuthorship W2019292712A5045414239 @default.
- W2019292712 hasAuthorship W2019292712A5078372604 @default.
- W2019292712 hasConcept C126322002 @default.
- W2019292712 hasConcept C126838900 @default.
- W2019292712 hasConcept C127413603 @default.
- W2019292712 hasConcept C141071460 @default.
- W2019292712 hasConcept C164705383 @default.
- W2019292712 hasConcept C2775841333 @default.
- W2019292712 hasConcept C2777798775 @default.
- W2019292712 hasConcept C2778333808 @default.
- W2019292712 hasConcept C2779450755 @default.
- W2019292712 hasConcept C2780007028 @default.
- W2019292712 hasConcept C2780645631 @default.
- W2019292712 hasConcept C2781156865 @default.
- W2019292712 hasConcept C500558357 @default.
- W2019292712 hasConcept C541997718 @default.
- W2019292712 hasConcept C71924100 @default.
- W2019292712 hasConcept C78519656 @default.
- W2019292712 hasConceptScore W2019292712C126322002 @default.
- W2019292712 hasConceptScore W2019292712C126838900 @default.
- W2019292712 hasConceptScore W2019292712C127413603 @default.
- W2019292712 hasConceptScore W2019292712C141071460 @default.
- W2019292712 hasConceptScore W2019292712C164705383 @default.
- W2019292712 hasConceptScore W2019292712C2775841333 @default.
- W2019292712 hasConceptScore W2019292712C2777798775 @default.
- W2019292712 hasConceptScore W2019292712C2778333808 @default.
- W2019292712 hasConceptScore W2019292712C2779450755 @default.
- W2019292712 hasConceptScore W2019292712C2780007028 @default.
- W2019292712 hasConceptScore W2019292712C2780645631 @default.
- W2019292712 hasConceptScore W2019292712C2781156865 @default.
- W2019292712 hasConceptScore W2019292712C500558357 @default.
- W2019292712 hasConceptScore W2019292712C541997718 @default.
- W2019292712 hasConceptScore W2019292712C71924100 @default.
- W2019292712 hasConceptScore W2019292712C78519656 @default.
- W2019292712 hasFunder F4320321001 @default.
- W2019292712 hasIssue "1-2" @default.