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- W2019333606 abstract "A role for non-protein sulfhydryl moieties (NPSH) (e.g., glutathione) in silica (SI)-induced cellular inflammation and fibrosis was examined in C57B1/6 mice depleted of lung NPSH by buthionine sulfoximine (BSO). Lung NPSH levels in the BSO-treated groups were reduced to approx. 50% of the non-BSO-treated animals. In BSO-treated Si-injected (2 mg/mouse) animals, the number of pulmonary alveolar macrophages (PAM) lavaged from the lungs was significantly increased on day 1 and decreased on day 7. Moreover, BSO-treated Si-exposed mice evidenced significantly more lavage protein and albumin on days 1 and 3, respectively, than non-BSO-treated Si-exposed mice. Si-induced collagen deposition, however, was decreased by 18% in the BSO-treated animals. These data suggest that lung NPSH lessens the potential of silica to elicit acute lung injury." @default.
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- W2019333606 date "1993-03-01" @default.
- W2019333606 modified "2023-09-23" @default.
- W2019333606 title "Modulation of silica-induced lung injury by reducing lung non-protein sulfhydryls with buthionine sulfoximine" @default.
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- W2019333606 doi "https://doi.org/10.1016/0378-4274(93)90012-m" @default.
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