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• W2019554647 abstract "To study the mechanism of curcumin-attenuated inflammation and liver pathology in early stage of alcoholic liver disease, female Sprague-Dawley rats were divided into four groups and treated with ethanol or curcumin via an intragastric tube for 4 weeks. A control group treated with distilled water, and an ethanol group was treated with ethanol (7.5 g/kg bw). Treatment groups were fed with ethanol supplemented with curcumin (400 or 1 200 mg/kg bw). The liver histopathology in ethanol group revealed mild-to-moderate steatosis and mild necroinflammation. Hepatic MDA, hepatocyte apoptosis, and NF-<mml:math xmlns:mml=http://www.w3.org/1998/Math/MathML><mml:mi>κ</mml:mi></mml:math>B activation increased significantly in ethanol-treated group when compared with control. Curcumin treatments resulted in improving of liver pathology, decreasing the elevation of hepatic MDA, and inhibition of NF-<mml:math xmlns:mml=http://www.w3.org/1998/Math/MathML><mml:mi>κ</mml:mi></mml:math>B activation. The 400 mg/kg bw of curcumin treatment revealed only a trend of decreased hepatocyte apoptosis. However, the results of SOD activity, PPAR<mml:math xmlns:mml=http://www.w3.org/1998/Math/MathML><mml:mi>γ</mml:mi></mml:math>protein expression showed no difference among the groups. In conclusion, curcumin improved liver histopathology in early stage of ethanol-induced liver injury by reduction of oxidative stress and inhibition of NF-<mml:math xmlns:mml=http://www.w3.org/1998/Math/MathML><mml:mi>κ</mml:mi></mml:math>B activation." @default.
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• W2019554647 date "2009-01-01" @default.
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• W2019554647 title "Curcumin Decreased Oxidative Stress, Inhibited NF-<mml:math xmlns:mml=http://www.w3.org/1998/Math/MathML><mml:mi>κ</mml:mi></mml:math>B Activation, and Improved Liver Pathology in Ethanol-Induced Liver Injury in Rats" @default.
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• W2019554647 doi "https://doi.org/10.1155/2009/981963" @default.
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