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• W2019574663 abstract "Modulation by sex hormones of aortic reactivity in rats with the metabolic syndrome (MS) was investigated. The following groups of weanling male Wistar rats were used: control rats (C) received regular tap water while MS rats received 30% sucrose in their drinking water; both had rodent chow for 24 weeks. These two groups were further subdivided into the following four groups: intact (Int), castrated (Cas), castrated plus testosterone (T) and castrated plus estradiol (E). Vascular response of thoracic aortic rings to norepinephrine (NE), acetylcholine (ACh), indomethacin (Indo) and nitro-l-arginine-methyl ester (l-NAME) was investigated. Blood pressure (BP) and serum nitrates and nitrites were measured. BP and serum nitrates and nitrites were modified by castration and treatments with either T or E. Vasoconstriction in Int MS and Cas MS + T aortas was larger than in C and Cas C + T, respectively. Vasodilation in Int MS and Cas MS + T was reduced in comparison with C and Cas C + T, Cas MS and Cas MS + E. Indomethacin decreased vasoconstriction in all groups (P < 0.002) but Int C and Cas C + T remained significantly smaller than Int MS and Cas MS + T. l-NAME in NE-contracted vessels induced a significant increase in vasoconstriction, except in Cas C + E rats; the responses of Int MS and Cas MS + T were significantly larger than in Int C and Cas C + T. The results suggest endothelial dysfunction in Int MS and Cas MS + T and a protective effect resulting from castration and castration plus E in MS animals, indicating a sex hormone influence." @default.
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• W2019574663 date "2007-05-01" @default.
• W2019574663 modified "2023-09-25" @default.
• W2019574663 title "Modulation of aortic vascular reactivity by sex hormones in a male rat model of metabolic syndrome" @default.
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• W2019574663 doi "https://doi.org/10.1016/j.lfs.2007.04.006" @default.
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