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• W2019903992 abstract "Retinoids bind to nuclear receptors [retinoic acid receptors (RARs) and retinoid X receptors]. RARβ, one of three isoforms of RARs (α, β, and γ), is expressed in the fetal and adult lung. We hypothesized that RARβ plays a role in alveolarization. Using morphometric analysis, we determined that there was a significant increase in the volume density of airspace in the alveolar region of the lung at 28, 42, and 56 d postnatal age in RARβ null mice when compared with wild-type controls. The mean cord length of the respiratory airspaces was increased in RARβ null animals at 42 d postnatal age. Respiratory gas-exchange surface area per unit lung volume was significantly decreased in RARβ null animals at 28, 42, and 56 d postnatal age. In addition, alveolar ducts tended to comprise a greater proportion of the lung airspaces in the RARβ null mice. The RARβ null mice also had impaired respiratory function when compared with wild-type control mice. There was no effect of RARβ gene deletion on lung platelet-derived growth factor (PDGF) receptor α mRNA levels in postnatal lung tissue at several postnatal ages. However PDGF-A protein levels were significantly lower in the RARβ null mice than in wild-type controls. Thus, deletion of the RARβ gene impairs the formation of the distal airspaces during the postnatal phase of lung maturation in mice via a pathway that may involve PDGF-A." @default.
• W2019903992 created "2016-06-24" @default.
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• W2019903992 date "2005-03-01" @default.
• W2019903992 modified "2023-10-05" @default.
• W2019903992 title "Alveolarization in Retinoic Acid Receptor-β–Deficient Mice" @default.
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• W2019903992 doi "https://doi.org/10.1203/01.pdr.0000151315.81106.d3" @default.
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