Matches in SemOpenAlex for { <https://semopenalex.org/work/W2019916416> ?p ?o ?g. }
Showing items 1 to 80 of
80
with 100 items per page.
- W2019916416 endingPage "398" @default.
- W2019916416 startingPage "387" @default.
- W2019916416 abstract "In succession to the previous quantitative work, a qualitative study has been carried out on the nature of a dual pathology affecting presynaptic boutons in the posterior tract nuclei of ageing rats. Based on the morphology of dystrophic boutons in early stage, it is concluded that the initial and therefore essential characteristic of dystrophic process is an abnormal increase of normal axonal components within the presynaptic boutons, and that various abnormal substructures of spheroids hitherto reported in the literature are probably the results of their secondary metamorphosis. The dystrophic process within the posterior tract nuclei is a selective one, involving presynaptic boutons and preterminal axons only of the posterior tract fibres. Comparison of the frequency of early dystrophic boutons and of fully grown-up spheroids indicates that a small percentage of boutons deriving from posterior tract fibres become dystrophic and of these dystrophic boutons only a small percentage again continue to develop unto large spheroids, throughout lifespan of the animals. On the other hand, in search of a morphological counterpart for the age-related decrease of volume ratio of presynaptic boutons to the neuropil, some dubious atrophic changes were also found in presynaptic boutons, which could have been easily missed from observation if studied qualitatively alone. Accordingly, no less numerous boutons other than dystrophic ones are supposed to atrophy 'independently' and to disappear 'silently' during the same period. The dystrophic and the atrophic changes involve different boutons (of different or the same terminal axons) within the same gray matter. This dual pathology of boutons needs further elucidation of its neurocytopathological as well as neurobiological background in the future." @default.
- W2019916416 created "2016-06-24" @default.
- W2019916416 creator A5013604848 @default.
- W2019916416 creator A5038532973 @default.
- W2019916416 date "1980-09-01" @default.
- W2019916416 modified "2023-10-16" @default.
- W2019916416 title "STUDY OF AXONAL DYSTROPHY. II. DYSTROPHY AND ATROPHY OF THE PRESYNAPTIC BOUTONS: A DUAL PATHOLOGY" @default.
- W2019916416 cites W1965764124 @default.
- W2019916416 cites W1966821569 @default.
- W2019916416 cites W1973868964 @default.
- W2019916416 cites W1977749841 @default.
- W2019916416 cites W1990597093 @default.
- W2019916416 cites W2002262944 @default.
- W2019916416 cites W2005003663 @default.
- W2019916416 cites W2011197036 @default.
- W2019916416 cites W2011529916 @default.
- W2019916416 cites W2015325297 @default.
- W2019916416 cites W2016327270 @default.
- W2019916416 cites W2021026048 @default.
- W2019916416 cites W2037778622 @default.
- W2019916416 cites W2084870443 @default.
- W2019916416 cites W2086116950 @default.
- W2019916416 cites W2104984365 @default.
- W2019916416 cites W2121427643 @default.
- W2019916416 cites W2124840810 @default.
- W2019916416 cites W2163686177 @default.
- W2019916416 cites W2328505992 @default.
- W2019916416 cites W2885183312 @default.
- W2019916416 cites W314350359 @default.
- W2019916416 cites W3143544522 @default.
- W2019916416 cites W4205296671 @default.
- W2019916416 doi "https://doi.org/10.1111/j.1365-2990.1980.tb00674.x" @default.
- W2019916416 hasPubMedId "https://pubmed.ncbi.nlm.nih.gov/7453943" @default.
- W2019916416 hasPublicationYear "1980" @default.
- W2019916416 type Work @default.
- W2019916416 sameAs 2019916416 @default.
- W2019916416 citedByCount "19" @default.
- W2019916416 crossrefType "journal-article" @default.
- W2019916416 hasAuthorship W2019916416A5013604848 @default.
- W2019916416 hasAuthorship W2019916416A5038532973 @default.
- W2019916416 hasConcept C105702510 @default.
- W2019916416 hasConcept C142724271 @default.
- W2019916416 hasConcept C169760540 @default.
- W2019916416 hasConcept C2777017193 @default.
- W2019916416 hasConcept C2777831001 @default.
- W2019916416 hasConcept C2781172350 @default.
- W2019916416 hasConcept C529278444 @default.
- W2019916416 hasConcept C71924100 @default.
- W2019916416 hasConcept C86803240 @default.
- W2019916416 hasConceptScore W2019916416C105702510 @default.
- W2019916416 hasConceptScore W2019916416C142724271 @default.
- W2019916416 hasConceptScore W2019916416C169760540 @default.
- W2019916416 hasConceptScore W2019916416C2777017193 @default.
- W2019916416 hasConceptScore W2019916416C2777831001 @default.
- W2019916416 hasConceptScore W2019916416C2781172350 @default.
- W2019916416 hasConceptScore W2019916416C529278444 @default.
- W2019916416 hasConceptScore W2019916416C71924100 @default.
- W2019916416 hasConceptScore W2019916416C86803240 @default.
- W2019916416 hasIssue "5" @default.
- W2019916416 hasLocation W20199164161 @default.
- W2019916416 hasLocation W20199164162 @default.
- W2019916416 hasOpenAccess W2019916416 @default.
- W2019916416 hasPrimaryLocation W20199164161 @default.
- W2019916416 hasRelatedWork W1978260998 @default.
- W2019916416 hasRelatedWork W1994517931 @default.
- W2019916416 hasRelatedWork W2019916416 @default.
- W2019916416 hasRelatedWork W2024541566 @default.
- W2019916416 hasRelatedWork W2065194738 @default.
- W2019916416 hasRelatedWork W2086051541 @default.
- W2019916416 hasRelatedWork W2158867611 @default.
- W2019916416 hasRelatedWork W2557505514 @default.
- W2019916416 hasRelatedWork W2889344046 @default.
- W2019916416 hasRelatedWork W2952689713 @default.
- W2019916416 hasVolume "6" @default.
- W2019916416 isParatext "false" @default.
- W2019916416 isRetracted "false" @default.
- W2019916416 magId "2019916416" @default.
- W2019916416 workType "article" @default.