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• W2019917449 startingPage "434" @default.
• W2019917449 abstract "Sinonasal polyposis (SNP) is a chronic inflammatory disease of nasal and paranasal cavities. Human leukocyte antigen-G molecules (HLA-G) are non-classic HLA-I molecules with anti-inflammatory and tolerogenic properties. HLA-G production is mainly induced by interleukin (IL)-10. IL-10 is an anti-inflammatory cytokine that inhibits the production of proinflammatory cytokines and induces HLA-class II down-modulation. Recent studies suggest that HLA-G could play a role in SNP pathogenesis; in SNP patients physiological levels of IL-10 (produced by activated peripheral blood CD14+ monocytes) are not able to induce production of HLA-G. Different mechanisms could justify these findings: genomic or amino-acidic sequence alterations in IL-10 lower IL-10 receptor expression, lower IL-10 receptor affinity, or alterations of the intracellular signal transmission. This study analyzes nucleotidic sequence of IL-10 gene in SNP patients. Sequencing of IL-10 gene shows that the lack of HLA-G production by peripheral blood CD14+ monocytes is not related to alterations in IL-10 gene nucleotidic sequence." @default.
• W2019917449 created "2016-06-24" @default.
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• W2019917449 date "2015-04-08" @default.
• W2019917449 modified "2023-09-26" @default.
• W2019917449 title "Analysis of Il-10 gene sequence in patients with sinonasal polyposis" @default.
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• W2019917449 doi "https://doi.org/10.1177/0394632015573922" @default.
• W2019917449 hasPubMedId "https://pubmed.ncbi.nlm.nih.gov/25875601" @default.
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