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- W2019925351 abstract "Adaptative left ventricular hypertrophy mainly derives from pressure or volume overload. Nevertheless, in the general population, about one-fifth of normotensives develop left ventricular hypertrophy despite a normal pressure load, while more than one-third of hypertensives do not develop it in response to pressure overload ( Table 1 ). It is unclear why some subjects become hypertrophic while others do not. Hypoxic or ischaemic myocyte loss could account for a limited number of such cases. The natural history of left ventricular hypertrophy is also very different, with some subjects developing heart failure and premature death and others who seem to be free of these prognostic effects1 ( Figure 1 ). Both left ventricular hypertrophy and its consequences are complex integrating multigenic traits acting in the long term.2 Figure 1 Cardiovascular and non-cardiovascular mortality rate (mean follow-up 19 ± 7 years) in 5896 subjects from the general population with and without left ventricular hypertrophy (left ventricular mass criteria or Minnesota code 3-1 or 3-3) (E. Casiglia et al. , unpublished data). NS, non-significant difference.View this table: Table 1 Prevalence of left ventricular hypertrophy (left ventricular mass index ≥125 g/m2 in men or ≥110 g/m2 in women) in 2368 subjects from the Italian general population (E. Casiglia et al. , unpublished data)It has been known since the 1990s that biochemical signalling events and changes in gene … *Corresponding author. Email: edoardo.casiglia{at}unipd.it" @default.
- W2019925351 created "2016-06-24" @default.
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- W2019925351 date "2008-03-19" @default.
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- W2019925351 title "Evolving concepts of left ventricular hypertrophy" @default.
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- W2019925351 doi "https://doi.org/10.1093/eurheartj/ehn055" @default.
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