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- W2019944950 abstract "SUMMARY SIN-1, the metabolite of molsidomine, caused a dose-dependent inhibition of the aggregation of rabbit platelets induced by adenosine diphosphate (ADP) and the stable thromboxane mimetic U-46619. Molsidomine was inactive in this respect. In the presence of a threshold concentration of prostacyclin, the antiaggregating activity of SIN-1 became more pronounced. As a result, the dose-response curve of SIN-1 was shifted to the left. These findings suggest that SIN-1 could be of therapeutic value to suppress platelet aggregation during atherosclerotic disease when the endogenous supply of endothelium-derived relaxing factor is compromised." @default.
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- W2019944950 date "1989-01-01" @default.
- W2019944950 modified "2023-09-27" @default.
- W2019944950 title "Interaction Between SIN-1 and Prostacyclin in Inhibiting Platelet Aggregation" @default.
- W2019944950 doi "https://doi.org/10.1097/00005344-198906152-00023" @default.
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