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- W2019987164 abstract "natural gas (predominantly methane, and small to trace References amounts of other hydrocarbons, nitrogen, carbon dioxide and mercapto compounds) are not known to have direct myocardial toxicity4 in the concentrations encountered; thus, hypoxia rather than toxicity must be considered the responsible mechanism.5 The hypoxic myocardial dysfunction in this patient was completely reversible, although in the interim an LV thrombus had formed rapidly and thus may complicate any form of acute cardiomyopathy. 1. Visaer CA, Kan 0, Lie KI, Durrer D. Incidence and one-year follow-up of left ventricular thrombus following acute myocardial infarction: an echocardiographic study of 96 patients (abstr). JACC 1983;1:648. 2. Wynne J, Braunwald E. The csrdiomyopathies and myocarditis. In: Braunwald E, ed. Heart Disease. A Texibook of Cardiovascula: Msdicine. W5 Seundcrs, 1984:1405-1406. 3. Braunwald E, Kroner RA. The stunned myocardium: prolonged postischemic ventricular dysfunction. Circulation 1982;66:1146-1149. 4. Casarett LJ, Doull J. Taxicology of the respiratory system. In: Casarett LJ, ed. Toxicology. The Basic Science of Poisons. New York: MacMillan, 19X:202-204. 5. Corya BC, Black MJ. McHenry PL. Echocardiographic findings after acute carbon monoxide poisoning. Br Heart J 1976;38:712-717." @default.
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- W2019987164 title "Massive endocardial calcification associated with endomyocardial fibrosis" @default.
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