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- W2020022583 abstract "Since the cross talk between inflammation and coagulation was described by Esmon et al. in 1991 [ 1 Esmon C.T. Taylor F.B. Snow T.R. Inflammation and coagulation linked processes potentially regulated through a common pathway mediated by protein C. Thromb Haemost. 1991; 66: 160-165 PubMed Google Scholar ], many vascular pathologies have appeared as complex disorders of the “vascular ecosystem,” which encompasses a fine-tuning of cellular events involving cells (parietal or circulating) and complex networks of soluble mediators. Leukocyte alterations are a cornerstone in the development of inflammatory conditions: They lead to activation of coagulation, microvascular plugging, and/or toxic attack of the vascular and perivascular environment, which are involved to different degrees in vascular pathologies [ 2 Freyburger G. Labrouche S. Flow cytometry assessment of leukocyte functions in vascular pathologies. Hematol Cell Ther. 1996; 38: 513-526 Crossref PubMed Scopus (9) Google Scholar ]. Leukocytes are easily withdrawn from circulating blood and are thus easier to study in clinical situations than parietal endothelial cells, although the functions of the latter are also important in regulation processes. A plethora of regulating soluble mediators (cytokines, chemokines, hormones, bioactive lipids, and serine proteases acting in an autocrine or paracrine manner) constitute a compact network of redundant regulating pathways. Most have been demonstrated in isolated in vitro systems, but their relative importance remains to be established in vivo." @default.
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- W2020022583 title "Influence of Hyperbaric Oxygen on Leukocyte Functions and Haemostasis in Normal Volunteer Divers" @default.
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- W2020022583 doi "https://doi.org/10.1016/s0049-3848(99)00107-3" @default.
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