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- W2020098765 endingPage "212" @default.
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- W2020098765 abstract "Activation of presynaptic receptors for a variety of neurotransmitters and neuromodulators inhibits transmitter release at many synapses. Such presynaptic inhibition might serve as a means of adjusting synaptic strength or preventing excessive transmitter release, or both. Previous evidence showed that presynaptic modulators inhibit Ca2+ channels and activate K+ channels at neuronal somata. These modulators also inhibit spontaneous transmitter release by mechanisms downstream of Ca2+ entry. The relative contribution of the above mechanisms to the inhibition of elicited release has been debated for a long time. Recent evidence at synapses where the relationship between transmitter release and presynaptic Ca2+ influx has been well characterized suggests that inhibition of presynaptic voltage-dependent Ca2+ channels plays the major role in presynaptic inhibition of elicited neurotransmitter release. In addition, modulation of the release machinery might contribute to inhibition of elicited release." @default.
- W2020098765 created "2016-06-24" @default.
- W2020098765 creator A5044806442 @default.
- W2020098765 creator A5066239290 @default.
- W2020098765 date "1997-05-01" @default.
- W2020098765 modified "2023-10-08" @default.
- W2020098765 title "Presynaptic inhibition of elicited neurotransmitter release" @default.
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- W2020098765 doi "https://doi.org/10.1016/s0166-2236(96)01015-6" @default.
- W2020098765 hasPubMedId "https://pubmed.ncbi.nlm.nih.gov/9141196" @default.
- W2020098765 hasPublicationYear "1997" @default.
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