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• W2020194124 endingPage "423" @default.
• W2020194124 startingPage "411" @default.
• W2020194124 abstract "Psychotic disorders, particularly schizophrenia, are associated with clinical phenomena that can be explained by disturbances in polyunsaturated fatty acid and prostaglandin metabolism. Previous studies of PUFA, PG synthesis, PGE1 receptor activity and aggregation responses in platelets, and clinical treatment trials suggest a role for PGE in the pathophysiology of schizophrenia. Since a decrease in PGE1 activity can be associated with an increase of dopamine release, a deficiency of PGE1 is consistent with the dopamine hypothesis of schizophrenia. State-of-the-art assay and clinical trial methodology should clarify the role of PUFA metabolism in schizophrenia." @default.
• W2020194124 created "2016-06-24" @default.
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• W2020194124 date "1989-07-01" @default.
• W2020194124 modified "2023-09-25" @default.
• W2020194124 title "Polyunsaturated Fatty Acids, Prostaglandins, and Schizophrenia" @default.
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• W2020194124 doi "https://doi.org/10.1111/j.1749-6632.1989.tb22626.x" @default.
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