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- W2020607517 abstract "Schizophrenic patients are extremely heavy tobacco smokers. However, a lower incidence of lung cancer in schizophrenic patients has been observed in comparison to other heavy smokers. Nicotine increases the proliferation of pulmonary neuroendocrine tissue, causing the release of a bombesin-like peptide. Thus, bombesin-like peptide levels in urine may be an indicator of precancerous, cigarette-induced lung damage. Bombesin-like peptide levels of 10 schizophrenic smokers and 11 schizophrenic nonsmokers were compared to those of nonschizophrenic subjects matched for age and pack-years of smoking. The nonschizophrenic smokers showed the expected increase in urinary bombesin-like peptide levels, as compared to nonschizophrenic nonsmokers. Schizophrenic patients had lower bombesin-like peptide levels independent of smoking effects. The mechanism of the difference in bombesin-like peptide levels between schizophrenic patients and nonschizophrenic subjects is unknown, but one possibility involves alteration in the α7-nicotinic acetylcholine receptor, which mediates the growth of some neuroendocrine cell lines in vitro." @default.
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- W2020607517 date "1999-01-01" @default.
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- W2020607517 title "Decreased Bombesin Peptide Response to Cigarette Smoking in Schizophrenia" @default.
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- W2020607517 doi "https://doi.org/10.1016/s0893-133x(98)00053-0" @default.
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