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• W2020751496 abstract "Mallory bodies (MBs) are characteristic of several liver disorders, and consist primarily of keratins with transglutaminase-generated keratin crosslinks. We tested the effect of the transglutaminase-2 (TG2) inhibitor KCC009 on MB formation in a mouse model fed 3,5-diethoxycarbonyl-1,4-dihydrocollidine (DDC). KCC009 decreased DDC-induced liver enlargement without affecting MB formation or extent of liver injury. TG2 protein and activity increased after DDC feeding and localized within and outside hepatocytes. KCC009 inhibited DDC-induced hepatomegaly by affecting hepatocyte cell size rather than proliferation. Hence, TG2 is a potential mediator of injury-induced hepatomegaly via modulation of hepatocyte hypertrophy, and KCC009-mediated TG2 inhibition does not affect mouse MB formation." @default.
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• W2020751496 date "2006-03-29" @default.
• W2020751496 modified "2023-10-17" @default.
• W2020751496 title "Pharmacologic transglutaminase inhibition attenuates drug-primed liver hypertrophy but not Mallory body formation" @default.
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• W2020751496 doi "https://doi.org/10.1016/j.febslet.2006.03.051" @default.
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