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• W2020775596 abstract "<h3>Abstract</h3> The involvement of the phytohormone ethylene as the main trigger of climacteric fruit ripening is well documented. However, our knowledge regarding the role of ethylene response factor (ERF) transcription factor in the transcriptional regulation of ethylene biosynthesis during fruit ripening remains limited. Here, comprehensive transcriptome analysis and expression profiling revealed 63 <i>ERF</i>s in durian pulps, termed <i>DzERF1</i>–<i>DzERF63</i>, of which 34 exhibited ripening-associated expression patterns at three stages (unripe, midripe, and ripe) during fruit ripening. Hierarchical clustering analysis classified 34 ripening-associated <i>DzERF</i>s into three distinct clades, among which, clade I consisted of downregulated <i>DzERF</i>s and clade III included those upregulated during ripening. Phylogenetic analysis predicted the functions of some DzERFs based on orthologs of previously characterized ERFs. Among downregulated <i>DzERF</i>s, DzERF6 functional prediction revealed its role as a negative regulator of ripening via ethylene biosynthetic gene repression, whereas among upregulated genes, DzERF9 was predicted to positively regulate ethylene biosynthesis. Correlation network analysis of 34 ripening-associated DzERFs with potential target genes revealed a strong negative correlation between DzERF6 and ethylene biosynthetic genes and a strong positive correlation between DzERF9 and ethylene biosynthesis. <i>DzERF6</i> and <i>DzERF9</i> showed differential expression patterns in association with different ripening treatments (natural, ethylene-induced, and 1-methylcyclopropene-delayed ripening). <i>DzERF6</i> was downregulated, whereas <i>DzERF9</i> was upregulated, during ripening and after ethylene treatment. The auxin-repressed and auxin-induced expression of <i>DzERF6</i> and <i>DzERF9</i>, respectively, confirmed its dose-dependent responsiveness to exogenous auxin. We suggest ethylene- and auxin-mediated roles of DzERF6 and DzERF9 during fruit ripening, possibly through transcriptional regulation of ethylene biosynthetic genes." @default.
• W2020775596 created "2016-06-24" @default.
• W2020775596 creator A5010515168 @default.
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• W2020775596 date "2004-05-01" @default.
• W2020775596 modified "2023-10-18" @default.
• W2020775596 title "The common SCN5A mutation R1193Q causes LQTS-type electrophysiological alterations of the cardiac sodium channel" @default.
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• W2020775596 doi "https://doi.org/10.1136/jmg.2003.013300" @default.
• W2020775596 hasPubMedCentralId "https://www.ncbi.nlm.nih.gov/pmc/articles/1618878" @default.
• W2020775596 hasPubMedId "https://pubmed.ncbi.nlm.nih.gov/15121794" @default.
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