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- W2021005768 abstract "A strong ornithine decarboxylase (ODC)-inactivating capacity has been previously shown (M.F. Zuretti and E. Gravela (1983) Biochim. Biophys. Acta, 742, 269-277) to be bound to rat liver microsomes. Present results show that in 2 fast-growing transplantable tumors, the 3924A Morris hepatoma and the AH 130 Yoshida ascites hepatoma, microsomes are endowed with a greatly enhanced ODC-inactivating capacity, and, concurrently, ODC displays an extreme in vitro liability and an unusual thiol-dependency (most of the activity requires dithiothreitol supply to be determined). These data are at variance with those previously obtained in hepatomas induced by N-2-fluorenylacetamide (E. Gravela et al., (1983) Cancer Res., 42, 2298-2300). The possibility that ODC liability in the 2 hepatomas here studied may result from in vivo exposure to a strong microsomal activity is considered." @default.
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- W2021005768 date "1987-07-01" @default.
- W2021005768 modified "2023-10-18" @default.
- W2021005768 title "Ornithine decarboxylase lability in 2 transplantable highly deviated rat hepatomas" @default.
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- W2021005768 doi "https://doi.org/10.1016/0304-3835(87)90100-5" @default.
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