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- W2021008851 abstract "Bone remodeling is a tightly regulated process of osteoclast-mediated bone resorption, balanced by osteoblast-mediated bone formation. Disruption of this balance can lead to increased bone turnover, resulting in excessive bone loss or extra bone formation and consequent skeletal disease. The receptor activator of nuclear factor kappaB ligand (RANKL) (along with its receptor), the receptor activator of nuclear factor kappaB and its natural decoy receptor, osteoprotegerin, are the final effector proteins of osteoclastic bone resorption. Here, I provide an overview of recent studies that highlight the key role of RANKL in the pathophysiology of several bone diseases and discuss the novel therapeutic approaches afforded by the modulation of RANKL." @default.
- W2021008851 created "2016-06-24" @default.
- W2021008851 creator A5080152292 @default.
- W2021008851 date "2009-03-01" @default.
- W2021008851 modified "2023-09-24" @default.
- W2021008851 title "Role of RANKL in bone diseases" @default.
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- W2021008851 doi "https://doi.org/10.1016/j.tem.2008.10.007" @default.
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