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- W2021018209 abstract "Although the problem of atrial fibrillation is now widely appreciated, the fundamental mechanisms that lead to arrhythmia onset and persistence have been difficult to elucidate. As a result, available pharmacologic therapies have focused more on modifying ion channel activity than on the underlying mechanisms. Recent studies suggest an important role for alterations in autonomic regulation, neurohormonal activation, and a systemic inflammatory state in the genesis and persistence of atrial fibrillation. The relative contributions of these distinct pathways to atrial fibrillation likely vary from patient to patient, and within a patient, as a function of age. Tailored therapies, together with patient-specific ablative interventions, may increase the success with which atrial fibrillation is treated and minimize the occurrence of life-threatening thromboembolic complications." @default.
- W2021018209 created "2016-06-24" @default.
- W2021018209 creator A5079314197 @default.
- W2021018209 date "2007-03-01" @default.
- W2021018209 modified "2023-09-25" @default.
- W2021018209 title "Recent Insights into the Pathophysiology of Atrial Fibrillation" @default.
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- W2021018209 doi "https://doi.org/10.1053/j.semtcvs.2007.01.006" @default.
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