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- W2021805882 endingPage "176" @default.
- W2021805882 startingPage "162" @default.
- W2021805882 abstract "All organisms rely on integrated networks to repair DNA double-strand breaks (DSBs) in order to preserve the integrity of the genetic information, to re-establish replication, and to ensure proper chromosomal segregation. Genetic, cytological, biochemical and structural approaches have been used to analyze how Bacillus subtilis senses DNA damage and responds to DSBs. RecN, which is among the first responders to DNA DSBs, promotes the ordered recruitment of repair proteins to the site of a lesion. Cells have evolved different mechanisms for efficient end processing to create a 3′-tailed duplex DNA, the substrate for RecA binding, in the repair of one- and two-ended DSBs. Strand continuity is re-established via homologous recombination (HR), utilizing an intact homologous DNA molecule as a template. In the absence of transient diploidy or of HR, however, two-ended DSBs can be directly re-ligated via error-prone non-homologous end-joining. Here we review recent findings that shed light on the early stages of DSB repair in Firmicutes." @default.
- W2021805882 created "2016-06-24" @default.
- W2021805882 creator A5001253974 @default.
- W2021805882 creator A5004838915 @default.
- W2021805882 creator A5005056186 @default.
- W2021805882 creator A5038388661 @default.
- W2021805882 creator A5046748617 @default.
- W2021805882 creator A5060299941 @default.
- W2021805882 date "2013-03-01" @default.
- W2021805882 modified "2023-10-01" @default.
- W2021805882 title "Early steps of double-strand break repair in Bacillus subtilis" @default.
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