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• W2021868503 abstract "Neuritin ( Nrn1 ) is a neurotrophin that plays an important role in nervous system plasticity and repair following nerve injury. MicroRNAs (miRNAs) are a type of small non‐coding RNA that regulate nearly all aspects of nerve development and survival, including apoptosis. Here it was found that miR‐204 negatively regulates Nrn1 protein expression through direct interaction with Nrn1 transcript. Moreover, miR‐204 activates cleaved caspase‐3, enhancing the sensitivity of RSC96 Schwann cells to H 2 O 2 ‐induced oxidative stress and apoptosis. Thus, miR‐204 expressed at a low level may create a microenvironment suitable for the repair of injured nerves by relieving the inhibition of Nrn1 transcription and stimulating the anti‐apoptotic function of Schwann cells. These results provide novel insights into the roles of miR‐204 in nerve injury and repair." @default.
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• W2021868503 date "2014-07-15" @default.
• W2021868503 modified "2023-10-15" @default.
• W2021868503 title "MiR‐204 promotes apoptosis in oxidative stress‐induced rat Schwann cells by suppressing <i>neuritin</i> expression" @default.
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• W2021868503 doi "https://doi.org/10.1016/j.febslet.2014.07.004" @default.
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