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- W2022142901 endingPage "138" @default.
- W2022142901 startingPage "129" @default.
- W2022142901 abstract "Erectile dysfunction (ED) is highly prevalent in diabetes mellitus. Pathophysiological mechanisms underlying diabetes-associated ED are in large part due to endothelial dysfunction, which functionally refers to the inability of the endothelium to produce vasorelaxing messengers and to maintain vasodilation and vascular homeostasis. The precise mechanisms leading to endothelial dysfunction in the diabetic vasculature, including the penis, are not yet fully understood. Hyperglycemia affects endothelial nitric oxide synthase activity and nitric oxide production/bioavailability, nitric oxide-independent relaxing factors, oxidative stress, production and/or action of hormones, growth factors and/or cytokines, and generation and activity of opposing vasoconstrictors. Considering recent advances in the field of vascular biology and diabetes, the emphasis in this review is placed on the mechanisms of hyperglycemia-induced endothelial dysfunction in the pathophysiology of diabetes-associated ED." @default.
- W2022142901 created "2016-06-24" @default.
- W2022142901 creator A5037019692 @default.
- W2022142901 creator A5084060127 @default.
- W2022142901 date "2006-06-15" @default.
- W2022142901 modified "2023-09-26" @default.
- W2022142901 title "Endothelial dysfunction in diabetic erectile dysfunction" @default.
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