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- W2022150984 endingPage "309" @default.
- W2022150984 startingPage "292" @default.
- W2022150984 abstract "Fanconi's syndrome (FS) was first described by Lignac in 1924. Associated with numerous varieties of inherited and acquired conditions, FS is characterized by a generalized transport defect in the proximal tubules, leading to renal losses of glucose, phosphate, calcium, uric acid, amino acids, bicarbonates, and other organic compounds. Cardinal symptoms of the syndrome are hyperaminoaciduria, glucosuria in the face of a normal serum glucose level, and phosphate wasting. Other symptoms may be associated, such as defects in bicarbonate reabsorption; renal acidification; urate reabsorption; urinary concentration; potassium conservation; reabsorption of sodium, calcium, and low-molecular-weight proteins; and secretion of p-aminohippuric acid. Acquired renal tubular defects resulting in FS also have been described in association with many exogenous agents, whether administered or accidentally ingested. This review concentrates on drug-induced FS." @default.
- W2022150984 created "2016-06-24" @default.
- W2022150984 creator A5017379947 @default.
- W2022150984 creator A5040938888 @default.
- W2022150984 creator A5070802786 @default.
- W2022150984 creator A5075352950 @default.
- W2022150984 date "2003-02-01" @default.
- W2022150984 modified "2023-09-25" @default.
- W2022150984 title "Drug-induced Fanconi's syndrome" @default.
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