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• W2022160113 endingPage "464" @default.
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• W2022160113 abstract "Activation of coagulation by tissue factor (TF) is frequently observed in sepsis syndrome and is documented in certain viral hemorrhagic fevers. Coagulation protease complexes signal by activating the G-protein coupled, protease-activated receptors that regulate inflammation. Blockade of TF attenuates lethality in experimental models of Ebola virus infection but – similar to findings in bacterial sepsis – reduction of inflammation, rather than attenuation of coagulation, predicts survival of treated animals. Thus, targeting TF appears to aid the antiviral immune response in hemorrhagic fevers, and further studies are encouraged to define how TF-dependent signaling regulates immunity. Activation of coagulation by tissue factor (TF) is frequently observed in sepsis syndrome and is documented in certain viral hemorrhagic fevers. Coagulation protease complexes signal by activating the G-protein coupled, protease-activated receptors that regulate inflammation. Blockade of TF attenuates lethality in experimental models of Ebola virus infection but – similar to findings in bacterial sepsis – reduction of inflammation, rather than attenuation of coagulation, predicts survival of treated animals. Thus, targeting TF appears to aid the antiviral immune response in hemorrhagic fevers, and further studies are encouraged to define how TF-dependent signaling regulates immunity." @default.
• W2022160113 created "2016-06-24" @default.
• W2022160113 creator A5005286957 @default.
• W2022160113 date "2004-09-01" @default.
• W2022160113 modified "2023-09-23" @default.
• W2022160113 title "Emerging roles of tissue factor in viral hemorrhagic fever" @default.
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• W2022160113 doi "https://doi.org/10.1016/j.it.2004.07.002" @default.
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