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- W2022162405 abstract "The immunological release of SRS-A was investigated in guinea-pig chopped lung. A number of unsaturated fatty acids, all of which are substrates for arachidonate lipoxygenase were found to potentiate the release of SRS-A. This potentiation was enhanced by indomethacin, a cyclo-oxygenase inhibitor, and completely reversed by nordihydroguaiaretic acid (NDGA) and eicosatetraynoic acid (ETA) which inhibit lipoxygenase. This suggests that some aspect of arachidonate lipoxygenase action stimulates release of SRS-A and that release of SRS-A is increased by redirection of arachidonic acid (AA) metabolism via the lipoxygenase pathway (Hamberg, 1976). However, although exogenous 14C-AA increased SRS-A output it was not incorporated into SRS-A." @default.
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- W2022162405 date "1980-03-01" @default.
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- W2022162405 title "The role of arachidonate lipoxygenase in the release of SRS-A from guinea-pig chopped lung" @default.
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- W2022162405 doi "https://doi.org/10.1016/0090-6980(80)90071-4" @default.
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