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• W2022167511 abstract "Recent examination of the many factors associated with the development of chronic rejection has led to the recognition that one of the important pathogenic mechanisms is responsible for the inflammatory and proliferative lesions seen in grafts that persist for extended periods of time. Clinical retrospective studies have provided evidence for an association between episodes of acute rejection and the appearance of antidonor antibodies with the development of the widespread vascular intimal proliferation and inflammatory changes of chronic rejection. Although insensitive enough to provide information on the importance of specific components of the host immune response for mediating chronic rejection, this data is consistent with a much larger and compelling base of experimental data directly addressing the nature of the host response to allografts with time. These studies in experimental animals have shown that: (1) the lesions of chronic rejection are largely the result of histocompatibility differences between donor and recipient; (2) multiple immune and nonimmune factors can influence the severity of the lesions; and (3) indirect donor antigen recognition and alloantibody production represent at least one important pathway for stimulating development of the lesions of chronic rejection." @default.
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• W2022167511 date "1998-10-01" @default.
• W2022167511 modified "2023-10-14" @default.
• W2022167511 title "The importance of humoral immune responses in chronic rejection" @default.
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• W2022167511 doi "https://doi.org/10.1016/s0955-470x(98)80007-4" @default.
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