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- W2022173379 abstract "Dicarbonyl stress is the abnormal accumulation of dicarbonyl metabolites leading to increased protein and DNA modification contributing to cell and tissue dysfunction in ageing and disease. Enzymes metabolising dicarbonyls, glyoxalase 1 and aldoketo reductases, provide an efficient and stress-response enzyme defence against dicarbonyl stress. Dicarbonyl stress is produced by increased formation and/or decreased metabolism of dicarbonyl metabolites, and by exposure to exogenous dicarbonyls. It contributes to ageing, disease and activity of cytototoxic chemotherapeutic agents." @default.
- W2022173379 created "2016-06-24" @default.
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- W2022173379 date "2015-03-01" @default.
- W2022173379 modified "2023-10-03" @default.
- W2022173379 title "Dicarbonyl stress in cell and tissue dysfunction contributing to ageing and disease" @default.
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- W2022173379 doi "https://doi.org/10.1016/j.bbrc.2015.01.140" @default.
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