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- W2022205209 endingPage "174" @default.
- W2022205209 startingPage "162" @default.
- W2022205209 abstract "Nuclear factor-κB (NF-κB) is a transcription factor that plays a crucial role in coordinating innate and adaptive immunity, inflammation, and apoptotic cell death. NF-κB is activated by various inflammatory stimuli including peptide factors and infectious microbes. It is also known as a redox-sensitive transcription factor activated by reactive oxygen species (ROS). Over the past decades, various investigators focused on the role of ROS in the activation of NF-κB by cytokines and lipopolysaccharides. However, recent studies also suggested that ROS have the potential to repress NF-κB activity. Currently, it is not well addressed how ROS regulate activity of NF-κB in a bidirectional fashion. In this paper, we summarize evidence for positive and negative regulation of NF-κB by ROS, possible redox-sensitive targets for NF-κB signaling, and mechanisms underlying biphasic and bidirectional influences of ROS on NF-κB, especially focusing on a role of ROS-mediated induction of endoplasmic reticulum stress." @default.
- W2022205209 created "2016-06-24" @default.
- W2022205209 creator A5058680290 @default.
- W2022205209 creator A5078775589 @default.
- W2022205209 date "2013-12-01" @default.
- W2022205209 modified "2023-10-01" @default.
- W2022205209 title "Bidirectional regulation of NF-κB by reactive oxygen species: A role of unfolded protein response" @default.
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