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• W2022227225 abstract "IL-1 receptor-associated kinase 4 (IRAK-4) is an effector of the Toll-like receptor and IL-1 receptor pathways that plays a critical role in innate immune responses. The role of IRAK-4 in adaptive immune functions in human subjects is incompletely understood.We sought to evaluate T-cell function in IRAK-4 deficient patients.We compared upregulation of CD25 and CD69 on T cells and production of IL-2, IL-6, and IFN-gamma after stimulation of PBMCs from 4 IRAK-4-deficient patients and healthy control subjects with anti-CD3 and anti-CD28.Upregulation of CD25 and CD69 on T cells and production of IL-6 and IFN-gamma, but not IL-2, was significantly reduced in IRAK-4-deficient patients.IRAK-4-deficient patients have defects in T-cell activation." @default.
• W2022227225 created "2016-06-24" @default.
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• W2022227225 date "2010-08-01" @default.
• W2022227225 modified "2023-10-14" @default.
• W2022227225 title "Impaired T-cell receptor activation in IL-1 receptor–associated kinase-4–deficient patients" @default.
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• W2022227225 doi "https://doi.org/10.1016/j.jaci.2010.05.013" @default.
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