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- W2022280655 abstract "The mechanisms underlying spontaneous remission of autoimmune diseases are presently unknown, though regulatory T cells are believed to play a major role in this process. We tested the hypothesis that Th2 and/or other T cell regulatory cytokines cause the spontaneous remission of experimental allergic encephalomyelitis (EAE), a model of Th1-mediated autoimmunity. We analyzed the cytokine profile of lymph node and central nervous system-infiltrating cells in individual SJL mice at different stages of proteolipid protein (PLP) 139-151 peptide-induced EAE. We found that IFN-gamma slowly fades away after clinical recovery, whereas IL-4, IL-10 and transforming growth factor-beta remain low or undetectable. Our peptide-results therefore suggest that regulatory T cells producing anti-inflammatory cytokines are not involved in spontaneous remission of EAE and challenge the view that the Th1/Th2 balance has a key role in EAE regulation." @default.
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- W2022280655 date "1998-12-01" @default.
- W2022280655 modified "2023-10-16" @default.
- W2022280655 title "Lack of Th2 cytokine increase during spontaneous remission of experimental allergic encephalomyelitis" @default.
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- W2022280655 doi "https://doi.org/10.1002/(sici)1521-4141(199812)28:12<3893::aid-immu3893>3.0.co;2-#" @default.
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