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• W2022295123 endingPage "178" @default.
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• W2022295123 abstract "Within the moderate exercise intensity domain, the speed of oxygen uptake (V̇O2) kinetics at the transition to a higher metabolic rate is thought to be limited by an inertia of the oxidative machinery. Nitric oxide (NO)-induced inhibition of O2 consumption within the electron transport chain may contribute to this inertia. This investigation tested the hypothesis that a reduction or removal of any such NO effect via infusion of Nω-nitro-l-arginine methyl ester (l-NAME; a NOS inhibitor) would speed V̇O2 kinetics at the onset of moderate exercise. Five Thoroughbred geldings underwent four transitions to running speeds of 7 m sec−1 (two control, C, 2 l-NAME [20 mg kg−1]) on an equine treadmill during which pulmonary gas exchange was determined using a bias flow system. Consistent with exercise in the moderate intensity domain, in none of the transitions was a V̇O2 slow component elicited. The l-NAME treatment significantly accelerated V̇O2 kinetics via a reduction of the primary amplitude time constant (C, 17.3±1.7; l-NAME, 11.8±1.5 sec, P<0.05) concomitant with faster overall dynamics (i.e. T50 and T75 both P<0.05) and a trend toward a decreased O2 deficit (C, 6.4±0.7; l-NAME, 4.7±1.2 L; P=0.06). These data support the notion that NO contributes prominently to the oxidative enzyme inertia and thus the speed of V̇O2 kinetics at the onset of moderate intensity exercise in the horse." @default.
• W2022295123 created "2016-06-24" @default.
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• W2022295123 date "2002-08-01" @default.
• W2022295123 modified "2023-10-15" @default.
• W2022295123 title "Nitric oxide synthase inhibition speeds oxygen uptake kinetics in horses during moderate domain running" @default.
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• W2022295123 doi "https://doi.org/10.1016/s1569-9048(02)00068-x" @default.
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