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• W2022361520 endingPage "691" @default.
• W2022361520 startingPage "686" @default.
• W2022361520 abstract "Vasculogenic mimicry (VM) refers to the unique ability of highly aggressive human tumor cells to form matrix-rich networks de novo when cultured on a three-dimensional matrix, thus mimicking embryonic vasculogenesis. Some studies have shown that tumor hypoxia can promote tumor cells to form vessel-like tubes in vitro and express genes associated with VM. Although, the mechanisms involved in hypoxia-induced VM remain elusive, we hypothesized that the epithelial–mesenchymal transition (EMT) regulator Twist may play a major role in hypoxia-induced VM. We investigated this hypothesis in vitro by pretreating hepatocellular carcinoma cells under hypoxic conditions. Following the hypoxia treatment, the cells formed typical pipe-like VM networks. Moreover, the expression of VM markers was increased. Hypoxia-induced VM was accompanied by the increased expression of Twist. Twist siRNA reversed the effects of hypoxia on VM. These results suggest that the overexpression of Twist correlates to hypoxia-induced VM in hepatocellular carcinoma cells." @default.
• W2022361520 created "2016-06-24" @default.
• W2022361520 creator A5002859742 @default.
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• W2022361520 date "2011-05-01" @default.
• W2022361520 modified "2023-10-08" @default.
• W2022361520 title "Role of Twist in vasculogenic mimicry formation in hypoxic hepatocellular carcinoma cells in vitro" @default.
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• W2022361520 doi "https://doi.org/10.1016/j.bbrc.2011.04.089" @default.
• W2022361520 hasPubMedId "https://pubmed.ncbi.nlm.nih.gov/21539816" @default.
• W2022361520 hasPublicationYear "2011" @default.
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