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- W2022365383 endingPage "1296" @default.
- W2022365383 startingPage "1283" @default.
- W2022365383 abstract "Plasmin, the principal downstream product of tissue-type plasminogen activator (tPA), is known for its potent fibrin-degrading capacity but is also recognized for many non-fibrinolytic activities. Curiously, plasmin has not been conclusively linked to blood–brain barrier (BBB) disruption during recombinant tPA (rtPA)-induced thrombolysis in ischemic stroke. This is surprising given the substantial involvement of tPA in the modulation of BBB permeability and the co-existence of tPA and plasminogen in both blood and brain throughout the ischemic event. Here, we review the work that argues a role for plasmin together with endogenous tPA or rtPA in BBB alteration, presenting the overall controversy around the topic yet creating a rational case for an involvement of plasmin in this process." @default.
- W2022365383 created "2016-06-24" @default.
- W2022365383 creator A5019155767 @default.
- W2022365383 creator A5084027827 @default.
- W2022365383 date "2014-06-04" @default.
- W2022365383 modified "2023-10-18" @default.
- W2022365383 title "Plasmin-Dependent Modulation of the Blood–Brain Barrier: A Major Consideration during tPA-Induced Thrombolysis?" @default.
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