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• W2022368603 abstract "1 The transport of ĤO−3 (Jsm) from a ĤO−3-buffered serosal to an unbuffered mucosal saline solution has been studied in rat caecum in vitro. 2 Carbachol, bethanechol and acetylcholine (ACh) caused a concentration-dependent fall in Jsm with similar maximum effects. 1,1-Dimethyl-4-phenyl-piperazinium iodide (DMPP) also inhibited Jsm but the effect was less than with the other drugs. Maximum cholinoceptor inhibition was less than that obtained with anoxia. 3 Responses were blocked by atropine (10−5 m) but hexamethonium (2 × 10−4 m) significantly altered the response only to DMPP. 4 Physostigmine (10−5 m) shifted the ACh response curve to the left but physostigmine itself caused inhibition of Jsm which was blocked by atropine. 5 Substitution of mucosal Cl− by NO−3 reduced Jsm to a similar extent to maximum cholinoceptor effect and abolished responses to bethanecol. Anoxia further reduced Jsm in the presence of NO−3. 6 Mucosal SITS and DIDS (1 mm) reduced Jsm but this was less than the maximum inhibition seen with drugs acting on cholinoceptors or mucosal Cl− removal. Serosal DIDS caused a similar inhibition. 7 We conclude that cholinoceptor agonists inhibit but do not abolish luminal bicarbonate transport by an action on muscarinic receptors." @default.
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• W2022368603 date "1991-06-01" @default.
• W2022368603 modified "2023-09-26" @default.
• W2022368603 title "The effect of drugs acting on cholinoceptors and mucosal chloride on luminal bicarbonate transport by rat caecum under in vitro conditions" @default.
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• W2022368603 doi "https://doi.org/10.1111/j.1476-5381.1991.tb09833.x" @default.
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