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- W2022387905 endingPage "516" @default.
- W2022387905 startingPage "507" @default.
- W2022387905 abstract "Endothelial dysfunction contributes to the pathogenesis of a variety of potentially serious infectious diseases and syndromes, including sepsis and septic shock, hemolytic-uremic syndrome, severe malaria, and dengue hemorrhagic fever. Because endothelial activation often precedes overt endothelial dysfunction, biomarkers of the activated endothelium in serum and/or plasma may be detectable before classically recognized markers of disease, and therefore, may be clinically useful as biomarkers of disease severity or prognosis in systemic infectious diseases. In this review, the current status of mediators of endothelial cell function (angiopoietins-1 and -2), components of the coagulation pathway (von Willebrand Factor, ADAMTS13, and thrombomodulin), soluble cell-surface adhesion molecules (soluble E-selectin, sICAM-1, and sVCAM-1), and regulators of vascular tone and permeability (VEGF and sFlt-1) as biomarkers in severe infectious diseases is discussed in the context of sepsis, E. coli O157:H7 infection, malaria, and dengue virus infection." @default.
- W2022387905 created "2016-06-24" @default.
- W2022387905 creator A5044405868 @default.
- W2022387905 creator A5089884077 @default.
- W2022387905 date "2013-08-15" @default.
- W2022387905 modified "2023-10-12" @default.
- W2022387905 title "Biomarkers of endothelial activation/dysfunction in infectious diseases" @default.
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