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- W2022396129 endingPage "597" @default.
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- W2022396129 abstract "Tissue injury and inflammation result in release of various mediators that promote ongoing pain or pain hypersensitivity against mechanical, thermal and chemical stimuli. Pro-nociceptive mediators activate primary afferent neurons directly or indirectly to enhance nociceptive signal transmission to the central nervous system. Excitation of primary afferents by peripherally originating mediators, so-called peripheral sensitization, is a hallmark of tissue injury-related pain. Many kinds of pro-nociceptive mediators, including ATP, glutamate, kinins, cytokines and tropic factors, synthesized at the damaged tissue, contribute to the development of peripheral sensitization. In the present review we will discuss the molecular mechanisms of peripheral sensitization following tissue injury." @default.
- W2022396129 created "2016-06-24" @default.
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- W2022396129 date "2013-11-01" @default.
- W2022396129 modified "2023-10-14" @default.
- W2022396129 title "Tissue Injury and Related Mediators of Pain Exacerbation" @default.
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- W2022396129 doi "https://doi.org/10.2174/1570159x11311060003" @default.
- W2022396129 hasPubMedCentralId "https://www.ncbi.nlm.nih.gov/pmc/articles/3849785" @default.
- W2022396129 hasPubMedId "https://pubmed.ncbi.nlm.nih.gov/24396335" @default.
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