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- W2022673246 abstract "Flow-induced vasodilatation may contribute to lowering of peripheral resistance to pregnancy. This study investigated modulation of flow responses by 17beta-estradiol.Small mesenteric arteries from prepubertal female Wistar rats were pretreated for 3 hours with 17beta-estradiol (10(-7) mol/L) with 17alpha-estradiol (10(-7) mol/L) or vehicle (0.1% dimethylsulfoxide). Responses to intraluminal flow were evaluated with use of a pressure arteriograph.After pretreatment with 17beta-estradiol arteries relaxed to flow, whereas those treated with vehicle or 17alpha-estradiol did not (percent change in diameter at maximum flow rate after 17beta-estradiol 38.7% +/- 5.7%, n = 10, vs 1.1% +/- 4.3%, n = 10 after vehicle; p < 0.01). Endothelium removal or pretreatment with either a nitric oxide synthase inhibitor or a novel soluble guanylyl cyclase inhibitor diminished the response to 17beta-estradiol.17Beta-estradiol stimulated nitric oxide-mediated flow-induced relaxation. Through this pathway 17beta-estradiol could play an important role in the control of vascular tone." @default.
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- W2022673246 date "1997-12-01" @default.
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- W2022673246 title "17β-Estradiol stimulates flow-induced vasodilatation in isolated small mesenteric arteries from prepubertal female rats" @default.
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- W2022673246 doi "https://doi.org/10.1016/s0002-9378(97)70087-5" @default.
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