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- W2022785083 endingPage "12" @default.
- W2022785083 startingPage "1" @default.
- W2022785083 abstract "Cell death mechanisms have been associated with the development of inflammatory bowel diseases in humans and mice. Recent studies suggested that a complex crosstalk between autophagy/apoptosis, microbe sensing, and enhanced endoplasmic reticulum stress in the epithelium could play a critical role in these diseases. In addition, necroptosis, a relatively novel programmed necrosis-like pathway associated with TNF receptor activation, seems to be also present in the pathogenesis of Crohn's disease and in specific animal models for intestinal inflammation. This review attempts to cover new data related to cell death mechanisms and inflammatory bowel diseases." @default.
- W2022785083 created "2016-06-24" @default.
- W2022785083 creator A5027011220 @default.
- W2022785083 creator A5053688988 @default.
- W2022785083 creator A5076177981 @default.
- W2022785083 date "2014-01-01" @default.
- W2022785083 modified "2023-10-17" @default.
- W2022785083 title "Cell Death and Inflammatory Bowel Diseases: Apoptosis, Necrosis, and Autophagy in the Intestinal Epithelium" @default.
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