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- W2022796087 abstract "HYPOTHYROIDISM causes depression of various enzymatic process and O2 consumption of the tissues (Boreld and Holmgreen, 1948; Tipton and Nixon, 1946). It also induces hypercholesterolemia, rise in liver cholesterol (Handler, 1948) and caracass cholesterol (Kennely and Maynard, 1952). A rise of volatile fatty acids (which include 90% acetic acid), acetone bodies and cholesterol with a depression of succinic and lactic dehydrogenase activity have been observed by the authors (1954) in the tissues of hypothyroid rats even when the effects of unequal food intake was neutralised. Acetate and acetoacetate are the basic materials for cholesterol synthesis (Brady and Gurin, 1951) and they are oxidized through the tricarboxylic acid cycle (Krebs, 1943; Krebs and Jhonson, 1937). Besides liver, kidney oxidises acetates very rapidly, forms acetoacetate (Medes et al., 1945a, 1945b) and also sjmthesises cholesterol (Srere, 1950). So all these facts make the metabolism of acetic acid a logical choice for study in liver a..." @default.
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- W2022796087 date "1955-05-01" @default.
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- W2022796087 title "METABOLISM OF ACETIC ACID IN KIDNEY AND LIVER SLICES OF THE HYPOTHYROID RATS" @default.
- W2022796087 doi "https://doi.org/10.1210/endo-56-5-507" @default.
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