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- W2022816420 endingPage "810" @default.
- W2022816420 startingPage "799" @default.
- W2022816420 abstract "The last decade has seen herpes simplex virus (HSV)-induced stromal keratitis (SK) research shift from being a topic only of interest to vision researchers to one that fascinates the general field of inflammatory disease. Studies on experimental mouse lesions have uncovered several fundamental processes that explain lesion development. In this model, the chronic immuno-inflammatory lesions are mainly orchestrated by CD4+ T cells, but multiple early events occur that set the stage for the subsequent pathology. These include virus replication, the production of key cytokines and chemokines, neovascularization of the avascular cornea and the influx of certain inflammatory cell types. Many of these early events are subject to modulation, providing an approach to controlling this important cause of human blindness. We also comment on events ongoing during chronic SK, debating whether or not these represent virus-induced or autoimmune lesions." @default.
- W2022816420 created "2016-06-24" @default.
- W2022816420 creator A5013287504 @default.
- W2022816420 creator A5035257973 @default.
- W2022816420 date "2005-04-01" @default.
- W2022816420 modified "2023-10-16" @default.
- W2022816420 title "Early events in HSV keratitis—setting the stage for a blinding disease" @default.
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