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- W2022896751 abstract "—The utilization of citrate by the cytoplasmic fraction of rat brain is inhibited in hypoxia and remains unaltered in anaesthesia. The addition of exogenous aspartate to the cytosolic fraction isolated from brains of hypoxic animals increases the rate of citrate removal. The level of cytosolic aspartate gradually decreases when the exposure period to low oxygen tension is increased and reaches a minimum after 30 min. The levels of mitochondrial aspartate and of cytoplasmic carbamyl aspartate remain constant. The low level of cytosolic aspartate is accompanied by an increase in the concentration of cytosolic urea and increase in the aspartate level in blood serum. It is suggested that the oxidation of citrate by the cytoplasmic fraction of brain is inhibited in hypoxia owing to the decrease in endogenous aspartate. The decrease in the level of cytoplasmic aspartate is caused by the diversion of this substrate toward urea synthesis and by the increased leakage across the cell/blood barrier to the blood stream. Anaesthesia prevents the changes induced by hypoxia." @default.
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- W2022896751 date "1975-10-01" @default.
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- W2022896751 title "THE EFFECT OF ASPARTATE ON CITRATE METABOLISM IN THE CYTOSOLIC FRACTION OF BRAIN UNDER CONDITIONS OF NORMOXIA, HYPOXIA AND ANAESTHESIA" @default.
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- W2022896751 doi "https://doi.org/10.1111/j.1471-4159.1975.tb04355.x" @default.
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