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• W2022951217 startingPage "217" @default.
• W2022951217 abstract "Bacteria evolve their capacity to cause disease by acquiring virulence genes that are usually clustered in discrete genetic modules termed pathogenicity islands (PAI). Stable integration of PAIs into pre-existing transcriptional networks coordinates expression from PAIs with ancestral genes in response to diverse environmental cues. Such transcriptional controls are evident in the regulation of the locus of enterocyte effacement (LEE), a PAI of enteropathogenic and enterohemorrhagic Escherichia coli. However, recent reports indicate that global post-transcriptional and post-translational regulators, including CsrA, Hfq and ClpXP, fine-tune the transcriptional output from the LEE. In this opinion article, we highlight recent advances in the understanding of post-transcriptional and post-translational regulation in attaching and effacing pathogens." @default.
• W2022951217 created "2016-06-24" @default.
• W2022951217 creator A5013818403 @default.
• W2022951217 creator A5046749543 @default.
• W2022951217 creator A5070554944 @default.
• W2022951217 date "2011-05-01" @default.
• W2022951217 modified "2023-09-26" @default.
• W2022951217 title "Honing the message: post-transcriptional and post-translational control in attaching and effacing pathogens" @default.
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• W2022951217 doi "https://doi.org/10.1016/j.tim.2011.01.004" @default.
• W2022951217 hasPubMedCentralId "https://www.ncbi.nlm.nih.gov/pmc/articles/3087826" @default.
• W2022951217 hasPubMedId "https://pubmed.ncbi.nlm.nih.gov/21333542" @default.
• W2022951217 hasPublicationYear "2011" @default.
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