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- W2023002208 endingPage "320" @default.
- W2023002208 startingPage "311" @default.
- W2023002208 abstract "Immune status is altered in patients with sepsis or non-infectious systemic inflammatory response syndrome (SIRS). Reduced ex-vivo TNF production by endotoxin-activated monocytes has been regularly reported. This observation is reminiscent of the phenomenon of endotoxin tolerance, and the term 'leukocyte reprogramming' well defines this phenomenon. This review will outline that the hyporesponsiveness of circulating leukocytes is not a generalized phenomenon in sepsis and SIRS. Indeed, the nature of the insult (i.e. infectious versus non-infectious SIRS; under anesthesia [surgery] or not [trauma, burn]), the nature of the activator used to trigger leukocytes (i.e. different Toll-like receptor ligands or whole bacteria), the nature of the cell culture (i.e. isolated monocytes versus peripheral blood mononuclear cells versus whole blood assays), and the nature of the analyzed cytokines (e.g. IL-1beta versus IL-1ra; TNF versus IL-10) have a profound influence on the outcome of the response." @default.
- W2023002208 created "2016-06-24" @default.
- W2023002208 creator A5036995113 @default.
- W2023002208 creator A5057372078 @default.
- W2023002208 creator A5063108232 @default.
- W2023002208 creator A5072038022 @default.
- W2023002208 date "2005-10-01" @default.
- W2023002208 modified "2023-09-25" @default.
- W2023002208 title "Reprogramming of circulatory cells in sepsis and SIRS" @default.
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