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• W2023004988 abstract "Significance The essential micronutrient zinc is known to modulate adaptive immune responses and dysregulated zinc homeostasis leads to immunodeficiency. However, the molecular mechanisms underlying this zinc-mediated modulation are unknown. We show that the zinc transporter ZIP10 plays an important role in B-cell receptor (BCR) signaling. Zip10 -deficiency in mature B cells attenuated both T-cell–dependent and –independent immune responses. Zip10 -deficient mature B cells proliferated poorly in response to BCR cross-linking, as a result of dysregulated BCR signaling. Our data establish that ZIP10 functions as a cellular regulator to modulate BCR signaling in humoral immune responses." @default.
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• W2023004988 date "2014-07-29" @default.
• W2023004988 modified "2023-09-27" @default.
• W2023004988 title "Zinc transporter SLC39A10/ZIP10 controls humoral immunity by modulating B-cell receptor signal strength" @default.
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• W2023004988 doi "https://doi.org/10.1073/pnas.1323557111" @default.
• W2023004988 hasPubMedCentralId "https://www.ncbi.nlm.nih.gov/pmc/articles/4136588" @default.
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• W2023004988 hasPublicationYear "2014" @default.
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