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- W2023014714 abstract "The electrical stimulation-induced responses of isolated dog thoracic ducts were investigated using an organ bath technique. Electrical stimulation (0.7 ms in pulse width, 25 V in nominal voltage, 10 s in duration time, 1–32 Hz at frequency) produced frequency-related contractions in the lymphatic preparations. The contractions were abolished by pretreatment with tetrodotoxin (10−7 M), guanethidine (10−7, 10−6 M), and bretylium (10−7, 10−6 M). Cocaine (10−6 M) significantly potentiated the electrical stimulation−induced contractions. Phentolamine (10−8–10−5 M), prazosin (10−8–10−5 M), bunazosin (10−6, 10−5 M), yohimbine (10−8–10−6 M) and rauwolscine (10−8–10−6 M) also dose−dependently reduced the contractions. On the other hand, propranolol (10−8–10−6 M), atropine (10−6 M), hexamethonium (10−6 M), aspirin (3×10−5 M), Nω-nitro-l-arginine methyl ester (L-NAME) (3×10−5 M) and L-NAME (3×10−5 M)+l-arginine (10−4 M) caused no significant effect on electrical stimulation-induced contractions. No significant difference in the electrical stimulation-induced responses was observed between the lymphatic preparations with and without an intact endothelium. The electrical stimulation caused only a small contraction with no relaxation in the thoracic duct preparation precontracted with 10−8 M U46619. The small contraction was abolished by 10−5 M phentolamine. These findings suggest that there exists α1- and α2-adrenoceptors-mediated excitatory innervation, but no NO-ergic inhibitory nerve fiber in dog thoracic ducts." @default.
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- W2023014714 date "1998-06-01" @default.
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- W2023014714 title "Electrical stimulation-induced α1- and α2-adrenoceptors-mediated contraction in isolated dog thoracic ducts" @default.
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- W2023014714 doi "https://doi.org/10.1016/s0165-1838(98)00057-5" @default.
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