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- W2023195670 endingPage "363" @default.
- W2023195670 startingPage "343" @default.
- W2023195670 abstract "The pathogenesis of type 2 diabetes is intimately intertwined with the vasculature. Insulin must efficiently enter the bloodstream from pancreatic β-cells, circulate throughout the body, and efficiently exit the bloodstream to reach target tissues and mediate its effects. Defects in the vasculature of pancreatic islets can lead to diabetic phenotypes. Similarly, insulin resistance is accompanied by defects in the vasculature of skeletal muscle, which ultimately reduce the ability of insulin and nutrients to reach myocytes. An underappreciated participant in these processes is the vascular pericyte. Pericytes, the smooth muscle-like cells lining the outsides of blood vessels throughout the body, have not been directly implicated in insulin secretion or peripheral insulin delivery. Here, we review the role of the vasculature in insulin secretion, islet function, and peripheral insulin delivery, and highlight a potential role for the vascular pericyte in these processes." @default.
- W2023195670 created "2016-06-24" @default.
- W2023195670 creator A5002389788 @default.
- W2023195670 creator A5042517821 @default.
- W2023195670 creator A5066344931 @default.
- W2023195670 date "2010-06-01" @default.
- W2023195670 modified "2023-10-04" @default.
- W2023195670 title "The Role of Blood Vessels, Endothelial Cells, and Vascular Pericytes in Insulin Secretion and Peripheral Insulin Action" @default.
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