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- W2023261925 abstract "To investigate the importance of progesterone (P4) metabolism by the 5alpha-reductase type I enzyme in mitigating P4 antiseizure effects.Ovariectomized, female homozygous and heterozygous 5alpha-reductase type I knockout mice (n = 23) and their wild-type siblings (n = 31) were administered P4 (1.0 mg), and their pentylenetetrazol (PTZ)-induced ictal behaviors were compared with those of vehicle-administered mice (n = 49).Mice deficient in the 5alpha-reductase type I enzyme administered P4, or vehicle-administered control mice, had significantly shorter latencies and increased incidence of PTZ-induced hindlimb extension and death than did wild-type mice administered P4.These data suggest that P4's metabolism by the 5alpha-reductase type I enzyme may mitigate some of P4's antiseizure effects in the PTZ-induced seizure model." @default.
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- W2023261925 date "2002-07-24" @default.
- W2023261925 modified "2023-09-30" @default.
- W2023261925 title "Progesterone Reduces Pentylenetetrazol-Induced Ictal Activity of Wild-Type Mice But Not Those Deficient in Type I 5α-Reductase" @default.
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- W2023261925 doi "https://doi.org/10.1046/j.1528-1157.43.s.5.19.x" @default.
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