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- W2023391636 endingPage "705" @default.
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- W2023391636 abstract "Human and animal studies suggest that there is a correlation between endogenous opioid peptides, especially beta-endorphin, and alcohol abuse. It has been proven that the consumption of alcohol activates the endogenous opioid system. Consumption of alcohol results in an increase in beta-endorphin level in those regions of the human brain, which are associated with a reward system. However, it has also been observed that habitual alcohol consumption leads to a beta-endorphin deficiency. It is a well-documented phenomenon that people with a genetic deficit of beta-endorphin peptide are particularly susceptible to alcoholism. The plasma level of beta-endorphin in subjects genetically at high risk of excessive alcohol consumption shows lower basal activity of this peptide. Its release increases significantly after alcohol consumption. Clinical and laboratory studies confirm that certain genetically determined factors might increase the individual's vulnerability to alcohol abuse." @default.
- W2023391636 created "2016-06-24" @default.
- W2023391636 creator A5008382424 @default.
- W2023391636 creator A5018370764 @default.
- W2023391636 date "2005-04-01" @default.
- W2023391636 modified "2023-09-29" @default.
- W2023391636 title "Deficit in beta-endorphin peptide and tendency to alcohol abuse" @default.
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- W2023391636 doi "https://doi.org/10.1016/j.peptides.2004.11.010" @default.
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