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• W2023408021 abstract "Microcystin-RR (MC-RR) has been suggested to induce apoptosis in tobacco BY-2 cells through mitochondrial dysfunction including the loss of mitochondrial membrane potential (ΔΨm). To further elucidate the mechanisms involved in MC-RR induced apoptosis in tobacco BY-2 cells, we have investigated the role of mitochondrial electron transport chain (ETC) as a potential source for reactive oxygen species (ROS). Tobacco BY-2 cells after exposure to MC-RR (60 mg/L) displayed apoptotic changes in association with an increased production of ROS and loss of ΔΨm. All of these adverse effects were significantly attenuated by ETC inhibitors including Rotenone (2 μmol/L, complex I inhibitor) and antimycin A (0.01 μmol/L, complex III inhibitor), but not by thenoyltrifluoroacetone (5 μmol/L, complex II inhibitor). These results suggest that mitochondrial ETC plays a key role in mediating MC-RR induced apoptosis in tobacco BY-2 cells through an increased mitochondrial production of ROS." @default.
• W2023408021 created "2016-06-24" @default.
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• W2023408021 date "2014-09-01" @default.
• W2023408021 modified "2023-09-28" @default.
• W2023408021 title "Mitochondrial electron transport chain is involved in microcystin-RR induced tobacco BY-2 cells apoptosis" @default.
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• W2023408021 doi "https://doi.org/10.1016/j.jes.2014.06.032" @default.
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