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- W2023455453 abstract "Diabetes mellitus is a common metabolic disorder associated with chronic complications including a state of mild to moderate cognitive impairment, in particular psychomotor slowing and reduced mental flexibility, not attributable to other causes, and shares many symptoms that are best described as accelerated brain ageing. A common theory for aging and for the pathogenesis of this cerebral dysfunctioning in diabetes relates cell death to oxidative stress in strong association to inflammation, and in fact nuclear factor κB (NFκB), a master regulator of inflammation and also a sensor of oxidative stress, has a strategic position at the crossroad between oxidative stress and inflammation. Moreover, metabolic inflammation is, in turn, related to the induction of various intracellular stresses such as mitochondrial oxidative stress, endoplasmic reticulum (ER) stress, and autophagy defect. In parallel, blockade of autophagy can relate to proinflammatory signaling via oxidative stress pathway and NFκB-mediated inflammation." @default.
- W2023455453 created "2016-06-24" @default.
- W2023455453 creator A5037690856 @default.
- W2023455453 creator A5085718869 @default.
- W2023455453 creator A5087243292 @default.
- W2023455453 creator A5087880161 @default.
- W2023455453 date "2014-01-01" @default.
- W2023455453 modified "2023-10-14" @default.
- W2023455453 title "Diabetes and the Brain: Oxidative Stress, Inflammation, and Autophagy" @default.
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